polyclonal rabbit anti phf2 antibody Search Results


90
Bio-Techne corporation phf2 antibody
Phf2 Antibody, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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93
Proteintech rabbit anti phf1 antibody
Rabbit Anti Phf1 Antibody, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
rabbit anti phf1 antibody - by Bioz Stars, 2026-08
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86
Novus Biologicals polyclonal rabbit anti phf2 antibody
Plant homeodomain finger 2 <t>(PHF2)</t> expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.
Polyclonal Rabbit Anti Phf2 Antibody, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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polyclonal rabbit anti phf2 antibody - by Bioz Stars, 2026-08
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phf2  (Bethyl)
94
Bethyl phf2
Plant homeodomain finger 2 <t>(PHF2)</t> expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.
Phf2, supplied by Bethyl, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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91
Proteintech rabbit anti phf2
Plant homeodomain finger 2 <t>(PHF2)</t> expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.
Rabbit Anti Phf2, supplied by Proteintech, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 91 stars, based on 1 article reviews
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Merck KGaA secondary antibody for β-actin
Plant homeodomain finger 2 <t>(PHF2)</t> expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.
Secondary Antibody For β Actin, supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
secondary antibody for β-actin - by Bioz Stars, 2026-08
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Santa Cruz Biotechnology anti c ebpα antibody
Clinicopathologic features of patients with ccRCC and correlation between nuclear <t> PHF2 </t> and <t> C/EBPα </t> expression and clinicopathologic parameters.
Anti C Ebpα Antibody, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Santa Cruz Biotechnology pkcε antibody
Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with <t>either</t> <t>GFP</t> or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of <t>PKCε</t> activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS
Pkcε Antibody, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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96
Proteintech tris buffered saline
Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with <t>either</t> <t>GFP</t> or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of <t>PKCε</t> activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS
Tris Buffered Saline, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
tris buffered saline - by Bioz Stars, 2026-08
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93
Proteintech npm
Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with <t>either</t> <t>GFP</t> or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of <t>PKCε</t> activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS
Npm, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/polyclonal+rabbit+anti+phf2+antibody/bio_rxiv__64898__2026__01__28__698016-72-33-34?v=Proteintech
Average 93 stars, based on 1 article reviews
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93
Proteintech whsc1
Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with <t>either</t> <t>GFP</t> or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of <t>PKCε</t> activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS
Whsc1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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DIAGENODE DIAGNOSTICS unspecific iggs
Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with <t>either</t> <t>GFP</t> or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of <t>PKCε</t> activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS
Unspecific Iggs, supplied by DIAGENODE DIAGNOSTICS, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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Image Search Results


Plant homeodomain finger 2 (PHF2) expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.

Journal: Journal of Pathology and Translational Medicine

Article Title: Implication of PHF2 Expression in Clear Cell Renal Cell Carcinoma

doi: 10.4132/jptm.2017.03.16

Figure Lengend Snippet: Plant homeodomain finger 2 (PHF2) expression in non-neoplastic renal tissue. PHF2 is expressed in the nucleus and cytoplasm of the proximal tubular epithelium and in the nucleus of the distal tubular epithelium and podocytes in the glomerulus. (A) Cortex. (B) Medulla.

Article Snippet: Polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO, USA) was diluted 1:200.

Techniques: Expressing

Relationship between  PHF2  expression and clinicopathological characteristics

Journal: Journal of Pathology and Translational Medicine

Article Title: Implication of PHF2 Expression in Clear Cell Renal Cell Carcinoma

doi: 10.4132/jptm.2017.03.16

Figure Lengend Snippet: Relationship between PHF2 expression and clinicopathological characteristics

Article Snippet: Polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO, USA) was diluted 1:200.

Techniques: Expressing, Biomarker Discovery

Kaplan-Meier curves. Cancer-specific and progression-free survival according to plant homeodomain finger 2 (PHF2) expression.

Journal: Journal of Pathology and Translational Medicine

Article Title: Implication of PHF2 Expression in Clear Cell Renal Cell Carcinoma

doi: 10.4132/jptm.2017.03.16

Figure Lengend Snippet: Kaplan-Meier curves. Cancer-specific and progression-free survival according to plant homeodomain finger 2 (PHF2) expression.

Article Snippet: Polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO, USA) was diluted 1:200.

Techniques: Expressing

Univariate and multivariate Cox regression analysis of cancer-specific and progression-free survival

Journal: Journal of Pathology and Translational Medicine

Article Title: Implication of PHF2 Expression in Clear Cell Renal Cell Carcinoma

doi: 10.4132/jptm.2017.03.16

Figure Lengend Snippet: Univariate and multivariate Cox regression analysis of cancer-specific and progression-free survival

Article Snippet: Polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO, USA) was diluted 1:200.

Techniques: Biomarker Discovery, Expressing

Clinicopathologic features of patients with ccRCC and correlation between nuclear  PHF2  and  C/EBPα  expression and clinicopathologic parameters.

Journal: Oncotarget

Article Title: The prognostic significance of nuclear expression of PHF2 and C/EBPα in clear cell renal cell carcinoma with consideration of adipogenic metabolic evolution

doi: 10.18632/oncotarget.19949

Figure Lengend Snippet: Clinicopathologic features of patients with ccRCC and correlation between nuclear PHF2 and C/EBPα expression and clinicopathologic parameters.

Article Snippet: Each polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO) and polyclonal rabbit anti-C/EBPα antibody (Santa Cruz Biotechnology, Dallas, TX) was diluted 1:200.

Techniques: Expressing

Multivariate analysis of cancer-specific and progression-free survival with  PHF2  and  C/EBPα  nuclear expression in 344 patients with ccRCC (Cox proportional hazard model).

Journal: Oncotarget

Article Title: The prognostic significance of nuclear expression of PHF2 and C/EBPα in clear cell renal cell carcinoma with consideration of adipogenic metabolic evolution

doi: 10.18632/oncotarget.19949

Figure Lengend Snippet: Multivariate analysis of cancer-specific and progression-free survival with PHF2 and C/EBPα nuclear expression in 344 patients with ccRCC (Cox proportional hazard model).

Article Snippet: Each polyclonal rabbit anti-PHF2 antibody (Novus Biologicals, Littleton, CO) and polyclonal rabbit anti-C/EBPα antibody (Santa Cruz Biotechnology, Dallas, TX) was diluted 1:200.

Techniques: Expressing

Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with either GFP or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of PKCε activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS

Journal: Nature Communications

Article Title: The histone demethylase Phf2 acts as a molecular checkpoint to prevent NAFLD progression during obesity

doi: 10.1038/s41467-018-04361-y

Figure Lengend Snippet: Liver-specific Phf2 overexpression causes quick-onset hepatosteatosis. a – h Mice, injected with either GFP or Phf2 overexpressing adenovirus, were studied 3 weeks later in the fed state. a Phf2 mice develop hepatic steatosis as shown by increased liver size and by oil red O staining of liver sections. Scale bars = 100 μm ( n = 10 per group). b Measurement of PKCε activity ( n = 6 per group). c Liver DAG content in the cytosol and at the plasma membrane ( n = 8 per group). d Relative expression of liver pro-inflammatory genes ( n = 10 per group). e Insulin tolerance test ( n = 10 per group). f Oral glucose tolerance test and insulin levels during the OGTT test ( n = 10 per group). g Western blot analysis of the PI3K/Akt signaling pathway in liver ( n = 20 per group). h Heatmap visualization of relative SFA, MUFA, and PUFA content in liver DAG, TG, and cholesterol ester species ( n = 10 per group). i – k Isolated primary hepatocytes overexpressing Phf2 and in which SCD1 expression was inhibited were incubated in the presence of palmitate (480 μM) for 24 h. i Representative western blots of the pro-inflammatory signaling pathway ( n = 4). j Percentage of hepatocyte SFA, MUFA and PUFA content in indicated culture conditions ( n = 3). k Representative western blot analysis of the PI3K/Akt signaling ( n = 4). All error bars represent mean ± SEM. Statistical analyses were made using unpaired t -test. * P < 0.01 GFP compared to Phf2, ** P < 0.05 GFP compared to Phf2. # P < 0.01 NCD compared to HFHS

Article Snippet: PKCε was immunoprecipitated from liver samples of GFP or Phf2 overexpressing mice using 0.5 μg of polyclonal rabbit anti PKCε antibody (Santa Cruz, CA).

Techniques: Over Expression, Injection, Staining, Activity Assay, Clinical Proteomics, Membrane, Expressing, Western Blot, Isolation, Incubation